Management with intravenous fluids can improve cardiac output in some surgical patients. Management with static preload indicators, such as central venous pressure and pulmonary artery occlusion pressure, has not demonstrated a suitable relationship with changes in the cardiac output induced by intravenous fluid therapy. Dynamic indicators, such as the variability of arterial pulse pressure or stroke volume variation, have demonstrated a suitable relationship. Since improvement in cardiac output does not guarantee an adequate perfusion pressure, in patients with hypotension, it is also necessary to know whether arterial pressure will also increase with intravenous fluid therapy. In this regard, the functional assessment of arterial load by dynamic arterial elastance could help to determine which patients will improve not only their cardiac output but also their mean arterial pressure.
MichardFBoussatSChemlaD, et al.Relation between respiratory changes in arterial pulse pressure and fluid responsiveness in septic. Am J Respir Crit Care Med. 2000;162(1):134–138.
2.
OsmanDRidelCRayP, et al.Cardiac filling pressures are not appropriate to predict hemodynamic response to volume challenge. Crit Care Med. 2007;35(1):64–68.
3.
WangPZhouMRanaMWBaZFChaudryIH. Differential alterations in microvascular perfusion in various organs during early and late sepsis. Am J Physiol. 1992;263(1 pt 1):G38–G43.
4.
ZhangLChenZDiaoYYangYFuP. Associations of fluid overload with mortality and kidney recovery in patients with acute kidney injury: a systematic review and meta-analysis. J Crit Care. 2015;30(4):860.e7–860.e13.
5.
SirventJ-MFerriCBaróAMurciaCLorencioC. Fluid balance in sepsis and septic shock as a determining factor of mortality. Am J Emerg Med. 2015;33(2):186–189.
6.
LeeJde LouwENiemiM, et al.Association between fluid balance and survival in critically ill patients. J Intern Med. 2015;277(4):468–477.
7.
UpadyaATilluckdharryLMuralidharanVAmoateng-AdjepongYManthousC. Fluid balance and weaning outcomes. Intensive Care Med. 2005;31(12):1643–1647.
8.
AlsousFKhamieesMDeGirolamoAAmoateng-AdjepongYManthousCA. Negative fluid balance predicts survival in patients with septic shock. Chest. 2000;117(6):1749–1754.
9.
YogendranSAsokumarBChengDCChungF. A prospective randomized double-blinded study of the effect of intravenous fluid therapy on adverse outcomes on outpatient surgery. Anesth Analg. 1995;80(4):682–686.
10.
VennRSteeleARichardsonPPolonieckiJGroundsMNewmanP. Randomized controlled trial to investigate influence of the fluid challenge on duration of hospital stay and perioperative morbidity in patients with hip fractures. Br J Anaesth. 2002;88(1):65–71.
11.
LopesMROliveiraMAPereiraVOSLemosIPBAulerJOCMichardF. Goal-directed fluid management based on pulse pressure variation monitoring during high-risk surgery: a pilot randomized controlled trial. Crit Care. 2007;11(5):R100.
12.
WakelingHGMcFallMRJenkinsCS, et al.Intraoperative oesophageal Doppler guided fluid management shortens postoperative hospital stay after major bowel surgery. Br J Anaesth. 2005;95(5):634–642.
13.
ChytraIPradlRBosmanRPelnárPKasalEZidkováA. Esophageal Doppler-guided fluid management decreases blood lactate levels in multiple-trauma patients: a randomized controlled trial. Crit Care. 2007;11(1):R24.
14.
SinclairSJamesSSingerM. Intraoperative intravascular volume optimisation and length of hospital stay after repair of proximal femoral fracture: randomised controlled trial. Br J Anaesth. 1997;315(7113):909–912.
15.
MagderS. How to use central venous pressure measurements. Curr Opin Crit Care. 2005;11(3):264–270.
16.
NahouraiiRARowellSE. Static measures of preload assessment. Crit Care Clin. 2010;26(2):295–305.
17.
CecconiMDe BackerDAntonelliM, et al.Consensus on circulatory shock and hemodynamic monitoring. Task Force of the European Society of Intensive Care Medicine. Intensive Care Med. 2014;40(12):1795–1815.
18.
MarikPEBaramMVahidB. Does central venous pressure predict fluid responsiveness? A systematic review of the literature and the tale of seven mares. Chest. 2008;134(1):172–178.
19.
MichardFTeboulJ. Predicting fluid responsiveness in ICU patients—a critical analysis of the evidence. Chest. 2002;121(6):2000–2008.
20.
DellingerRPCarletJMMasurH, et al.Surviving sepsis campaign guidelines for management of severe sepsis and septic shock. Crit Care Med. 2004;32(3):858–873.
21.
HollenbergSMAhrensTSAnnaneD, et al.Practice parameters for hemodynamic support of sepsis in adult patients: 2004 update. Crit Care Med. 2004;32(9):1928–1948.
22.
RhodesAEvansLEAlhazzaniW, et al.Surviving sepsis campaign. Crit Care Med. 2017;43(3):304–377.
23.
DiebelLNWilsonRFTagettMGKlineRA. End-diastolic volume. A better indicator of preload in the critically ill. Arch Surg. 1992;127(7):817–821; discussion 821-822.
24.
DiebelLWilsonRFHeinsJLarkyHWarsowKWilsonS. End-diastolic volume versus pulmonary artery wedge pressure in evaluating cardiac preload in trauma patients. J Trauma. 1994;37(6):950–955.
25.
WagnerJGLeathermanJW. Right ventricular end-diastolic volume as a predictor of the hemodynamic response to a fluid challenge. Chest. 1998;113(4):1048–1054.
26.
ThysDMHillelZGoldmanMMindichBPKaplanJA. A comparison of hemodynamic indices derived by invasive monitoring and two-dimensional echocardiography. Anesthesiology. 1987;67(5):630–634.
27.
CheungATSavinoJSWeissSJAukburgSJBerlinJA. Echocardiographic and hemodynamic index of left ventricular preload in patients with normal and abnormal ventricular funtion. Anesthesiology. 1994;81(2):376–387.
28.
SwensonJDHarkinCPaceNLAstleKPaceLBaileyP.Transesophageal echocardiography: an objective tool in defining maximum ventricular response to intravenous fluid therapy. Anesth Analg. 1996;83(6):1149–1153.
29.
TousignantCPWalshFMazerCD. The use of transesophageal echocardiography for preload assessment in critically ill patients. Anesth Analg. 2000;90(2):351–355.
30.
TarvernierBMakhotineOLebuffeGDupontJScherpereelP. Systolic pressure variation asa a guide to fluid therapy in patients with sepsis-inducuced hypotension. Anesthesiology. 1998;89(6):1313–1321.
31.
FeisselMMichardFManginIRuyerOFallerJTeboulJ-L. Respiratory changes in aortic blood velocity as an indicator of fluid responsiveness in ventilated patients with septic shock. Chest. 2001;119(3):867–873.
32.
BerlinDABakkerJ. Starling curves and central venous pressure starling curves and central venous pressure. Crit Care. 2015;19(1):55.
33.
ReuterDKirchnerAFelbingerTW, et al.Usefulness of left ventricular stroke volume variation to assess fluid responsiveness in patients with reduced cardiac function. Crit Care Med. 2003;31(5):1399–1404.
34.
BerkenstadtHMargalitNHadaniM, et al.Stroke volume variation as a predictor of fluid responsiveness in patients undergoing brain surgery. Anesth Analg. 2001;92(4):984–989.
35.
MonnetXRienzoMOsmanD, et al.Esophageal doppler monitoring predicts fluid responsiveness in critically ill ventilated patients. Intensive Care Med. 2005;31(9):1195–1201.
36.
MyatraSNPrabuNRDivatiaJVMonnetXKulkarniAPTeboulJ. The changes in pulse pressure variation or stroke volume variation after a “tidal volume challenge” reliably predict fluid responsiveness during low tidal volume ventilation. Crit Care Med. 2017;(45):415–421.
37.
BendjelidKSuterPMRomandJA. The respiratory change in preejection period: a new method to predict fluid responsiveness. J Appl Physiol. 2004;96(1):337–342.
38.
FeisselMTeboulJLMerlaniPBadieJFallerJPBendjelidK. Plethysmographic dynamic indices predict fluid responsiveness in septic ventilated patients. Intensive Care Med. 2007;33(6):993–999.
39.
Vieillard-BaronACherguiKRabillerA, et al.Superior vena caval collapsibility as a gauge of volume status in ventilated septic patients. Intensive Care Med. 2004;30(9):1734–1739.
40.
BarbierCLoubièresYSchmitC, et al.Respiratory changes in inferior vena cava diameter are helpful in predicting fluid responsiveness in ventilated septic patients. Intensive Care Med. 2004;30(9):1740–1746.
41.
MagderSGeorgiadisGCheongT. Respiratory variations in right atrial pressure predict the response to fluid challenge. J Crit Care. 1992;7(2):76–85.
42.
MonnetXOsmanDRidelCLamiaBRichardCTeboulJ-L. Predicting volume responsiveness by using the end-expiratory occlusion in mechanically ventilated intensive care unit patients. Crit Care Med. 2009;37(3):951–956.
43.
PerelAMinkovichLPreismanSAbiadMSegalECoriatP. Assessing fluid-responsiveness by a standardized ventilatory maneuver: the respiratory systolic variation test. Anesth Analg. 2005;100(4):942–945.
44.
PreismanSKoganSBerkenstadtHPerelA. Predicting fluid responsiveness in patients undergoing cardiac surgery: functional haemodynamic parameters including the respiratory systolic variation test and static preload indicators. Br J Anaesth. 2005;95(6):746–755.
45.
LafanechèreAPèneFGoulenokC, et al.Changes in aortic blood flow induced by passive leg raising predict fluid responsiveness in critically ill patients. Crit Care. 2006;10(5):1–8.
46.
Monge GarcíaMIGil CanoADíaz MonrovéJC. Arterial pressure changes during the valsalva maneuver to predict fluid responsiveness in spontaneously breathing patients. Intensive Care Med. 2009;35(1):77–84.
47.
IgnacioMGarcíaMCanoAGRomeroMGPintadoRMMadueñoVP. Non-invasive assessment of fluid responsiveness by changes in partial end-tidal CO2 pressure during a passive leg-raising maneuver. Ann Intensive Care. 2012;2(1):9.
48.
TusmanGGroismanIMaidanaGA, et al.The sensitivity and specificity of pulmonary carbon dioxide elimination for noninvasive assessment of fluid responsiveness. Anesth Analg. 2016;122(5):1404–1411.
49.
MullerLToumiMBousquetP-J, et al.An increase in aortic blood flow after an infusion of 100 ml colloid over 1 minute can predict fluid responsiveness. Anesthesiology. 2011;115(3):541–547.
50.
CavallaroFSandroniCAntonelliM. Functional hemodynamic monitoring and dynamic indices of fluid responsiveness. Minerva Anestesiol. 2008;74(4):123–135.
51.
RickJJBurkeSS. Respirator paradox. South Med J. 1978;71(11):1376–8, 1382.
52.
PerelAPizovRCotevS. Systolic blood pressure variation is a sensitive indicator of hypovolemia in ventilated dogs subjected to graded hemorrahge. Anesthesiology. 1987;67(4):498–502.
53.
PizovRYa'ariYPerelA. The arterial pressure waveform during acute ventricular failure and synchronized external chest compression. Anesth Analg. 1989;68(2):150–156.
54.
DenaultAYGasiorTAGorcsanJIIIMandarinoWADeneaultLGPinskyMR. Determinants of aortic pressure variation during positive-pressure ventilation in man. Chest. 1999;116(1):176–186.
55.
BeaussierMCoriatPPerelA, et al.Determinants of systolic pressure variation in patients ventilated after vascular surgery. J Cardiothorac Vasc Anesth. 1995;9(5):547–551.
56.
ReuterDBayerleinJGoepfertMSG, et al.Influence of tidal volume on left ventricular stroke volume variation measured by pulse contour analysis in mechanically ventilated patients. Intensive Care Med. 2003;29(3):476–480.
57.
ChemlaDHébertJLCoiraultC, et al.Total arterial compliance estimated by stroke volume-to-aortic pulse pressure ratio in humans. Am J Physiol. 1998;274(2 pt 2):H500–H505.
58.
MonnetXBleibtreuAFerréA, et al.Passive leg-raising and end-expiratory occlusion tests perform better than pulse pressure variation in patients with low respiratory system compliance. Crit Care Med. 2012;40(1):152–157.
59.
MichardFChemlaDRichardC, et al.Clinical use of respiratory changes in arterial pulse pressure to monitor the hemodynamic effects of PEEP. Am J Respir Crit Care Med. 1999;159(3):935–939.
60.
De BackerDHeenenSPiagnerelliMKochMVincentJ-L. Pulse pressure variations to predict fluid responsiveness: influence of tidal volume. Intensive Care Med. 2005;31(4):517–523.
61.
MullerLLouartGBousquetP-J, et al.The influence of the airway driving pressure on pulsed pressure variation as a predictor of fluid responsiveness. Intensive Care Med. 2010;36(3):496–503.
62.
ValléeFRichardJCMMariA, et al.Pulse pressure variations adjusted by alveolar driving pressure to assess fluid responsiveness. Intensive Care Med. 2009;35(6):1004–1010.
63.
SlamaMMaizelJ.Pulse pressure variations in acute respiratory distress syndrome: “fifty shades of grey.”Crit Care Med. 2016;44(2):452–453.
LiuYWeiLLiGYuXLiGLiY. Pulse pressure variation adjusted by respiratory changes in pleural pressure, rather than by tidal volume, reliably predicts fluid responsiveness in patients with acute respiratory distress syndrome. Crit Care Med. 2016;44(2):342–351.
66.
De BackerDTacconeFSHolstenRIbrahimiFVincentJ-L. Influence of respiratory rate on stroke volume variation in mechanically ventilated patients. Anesthesiology. 2009;110(5):1092–1097.
67.
MahjoubYPilaCFriggeriA, et al.Assessing fluid responsiveness in critically ill patients: false-positive pulse pressure variation is detected by doppler echocardiographic evaluation of the right ventricle. Crit Care Med. 2009;37(9):2570–2575.
68.
KimYKShinWJSongJG, et al.Effect of right ventricular dysfunction on dynamic preload indices to predict a decrease in cardiac output after inferior vena cava clamping during liver transplantation. Transplant Proc. 2010;42(7):2585–2589.
69.
Wyler von BallmoosMTakalaJRoeckM, et al.Pulse-pressure variation and hemodynamic response in patients with elevated pulmonary artery pressure: a clinical study. Crit Care. 2010;14(3):R111.
70.
MonnetXRienzoMOsmanD, et al.Passive leg raising predicts fluid responsiveness in the critically ill. Crit Care Med. 2006;34(5):1402–1407.
71.
CannessonMLe ManachYHoferCK, et al.Assessing the diagnostic accuracy of pulse pressure variations for the prediction of fluid responsiveness: a ‘gray zone’ approach. Anesthesiology. 2011;89(2):231–241.
72.
BiaisMEhrmannSMariA, et al.Clinical relevance of pulse pressure variations for predicting fluid responsiveness in mechanically ventilated intensive care unit patients: the grey zone approach. Crit Care. 2014;18(6):587.
73.
PernerAFaberT. Stroke volume variation does not predict fluid responsiveness in patients with septic shock on pressure support ventilation. Acta Anaesthesiol Scand. 2006;50(9):1068–1073.
74.
MullerLBobbiaXToumiM, et al.Respiratory variations of inferior vena cava diameter to predict fluid responsiveness in spontaneously breathing patients with acute circulatory failure: need for a cautious use. Crit Care. 2012;16(5):R188.
75.
AirapetianNMaizelJAlyamaniO, et al.Does inferior vena cava respiratory variability predict fluid responsiveness in spontaneously breathing patients?Crit Care. 2015;19(1):400.
76.
MahjoubYTouzeauJAirapetianN, et al.The passive leg-raising maneuver cannot accurately predict fluid responsiveness in patients with intra-abdominal hypertension. Crit Care Med. 2010;38(9):1824–1829.
77.
ShimJ-KSongJ-WSongYKimJ-HKangH-MKwakY-L. Pulse pressure variation is not a valid predictor of fluid responsiveness in patients with elevated left ventricular filling pressure. J Crit Care. 2014;29(6):987–991.
78.
HeenenSDe BackerDVincentJ-L. How can the response to volume expansion in patients with spontaneous respiratory movements be predicted?Crit Care. 2006;10(4):R102.
79.
BerkenstadtHFriedmanZPreismanSKeidanILivingstoneDPerelA. Pulse pressure and stroke volume variations during severe haemorrhage in ventilated dogs. Br J Anaesth. 2005;94(6):721–726.
80.
ValtierBCholleyBPBelotJDe CoussayeJMateoJPayenDM. Noninvasive monitoring of cardiac output in critically ill patients using transesophageal doppler. Am J Respir Crit Care Med. 1998;158(1):77–83.
81.
RoeckMJakobSMBoehlenTBranderLKnueselRTakalaJ. Change in stroke volume in response to fluid challenge: assessment using esophageal doppler. Intensive Care Med. 2003;29(10):1729–1735.
82.
DarkPMSingerM. The validity of trans-esophageal Doppler ultrasonography as a measure of cardiac output in critically ill adults. Intensive Care Med. 2004;30(11):2060–2066.
83.
WallaceAGMitchellJHSkinnerNSSarnoffSJ. Duration of the phases of left ventricular systole. Circ Res. 1963;12(6):611–619.
84.
BrundinTHedenstiernaGMcCarthyG. Effect of intermittent positive pressure ventilation on cardiac systolic time intervals. Acta Anaesthesiol Scand. 1976;20(4):278–284.
85.
FeisselMBadieJMerlaniPGFallerJ-PBendjelidK. Pre-ejection period variations predict the fluid responsiveness of septic ventilated patients. Crit Care Med. 2005;33(11):2534–2539.
86.
PartridgeBL. Use of pulse oximetry as a noninvasive indicator of intravascular volume status. J Clin Monit. 1987;3(4):263–268.
87.
ShamirMEidelmanLAFlomanYKaplanLPizovR. Pulse oximetry plethysmographic waveform during changes in blood volume. Br J Anaesth. 1999;82(2):178–181.
88.
NataliniGRosanoATarantoMFaggianBVittorielliEBernardiniA. Arterial versus plethysmographic dynamic indices to test responsiveness for testing fluid administration in hypotensive patients: a clinical trial. Anesth Analg. 2006;103(6):1478–1484.
89.
CannessonMBesnardCDurandPGBohéJJacquesD. Relation between respiratory variations in pulse oximetry plethysmographic waveform amplitude and arterial pulse pressure in ventilated patients. Crit Care. 2005;9(5):R562–R568.
90.
LandsverkSAHoisethLOKvandalPHisdalJSkareOKirkeboenKA. Poor agreement between respiratory variations in pulse oximetry photoplethysmographic waveform amplitude and pulse pressure in intensive care unit patients. Anesthesiology. 2008;109(5):849–855.
91.
GolparvarMNaddafniaHSaghaeiM. Evaluating the relationship between arterial blood pressure changes and indices of pulse oximetric plethysmography. Anesth Analg. 2002;95(6):1686–1690.
92.
GuytonACLindseyAWAbernathyBRichardsonT. Venous return at various right atrial pressures and the normal venous return curve. Am J Physiol. 1957;189(3):609–615.
93.
MorganBMartinWHornbeinTCrawfordEGuntherothW. Hemodynamic effects of intermittent positive pressure respiration. Anesthesiology. 1966;27(5):584–590.
94.
van den BergPCMJansenJRCPinskyMR. Effect of positive pressure on venous return in volume-loaded cardiac surgical patients. J Appl Physiol. 2002;92(3):1223–1231.
95.
AmooreJNSantamoreWP. Venous collapse and the respiratory variability in systemic venous return. Cardiovasc Res. 1994;28(4):472–479.
96.
Vieillard-BaronAAugardeRPrinSPageBBeauchetAJardinF. Influence of superior vena caval zone condition on cyclic changes in right ventricular outflow during respiratory support. Anesthesiology. 2001;95(5):1083–1088.
97.
JardinFFarcotJCGueretPProstJFOzierYBourdariasJP. Cyclic changes in arterial pulse during respiratory support. Circulation. 1983;68(2):266–274.
98.
FeisselMMichardFFallerJ-PTeboulJ-L. The respiratory variation in inferior vena cava diameter as a guide to fluid therapy. Intensive Care Med. 2004;30(9):1834–1837.
99.
GuiottoGMasaroneMPaladinoF, et al.Inferior vena cava collapsibility to guide fluid removal in slow continuous ultrafiltration: a pilot study. Intensive Care Med. 2010;36(4):692–696.
100.
CheriexECLeunissenKMJanssenJHMooyJMvan HooffJP. Echography of the inferior vena cava is a simple and reliable tool for estimation of “dry weight” in haemodialysis patients. Nephrol Dial Transplant. 1989;4(6):563–568.
101.
BrennanJMRonanAGoonewardenaS, et al.Handcarried ultrasound measurement of the inferior vena cava for assessment of intravascular volume status in the outpatient hemodialysis clinic. Clin J Am Soc Nephrol. 2006;1(4):749–753.
102.
BrennanJMBlairJEGoonewardenaS, et al.Reappraisal of the use of inferior vena cava for estimating right atrial pressure. J Am Soc Echocardiogr. 2007;20(7):857–861.
103.
NagdevADMerchantRCTirado-GonzalezASissonCAMurphyMC. Emergency department bedside ultrasonographic measurement of the caval index for noninvasive determination of low central venous pressure. Ann Emerg Med. 2010;55(3):290–295.
104.
ArthurMELandolfoCWadeMCastresanaMR. Inferior vena cava diameter (IVCD) measured with transesophageal echocardiography (TEE) can be used to derive the central venous pressure (CVP) in anesthetized mechanically ventilated patients. Echocardiography. 2009;26(2):140–149.
105.
JueJChungWSchillerNB. Does inferior vena cava size predict right atrial pressures in patients receiving mechanical ventilation?J Am Soc Echocardiogr. 1992;5(6):613–619.
106.
VignonP. Evaluation of fluid responsiveness in ventilated septic patients: back to venous return. Intensive Care Med. 2004;30(9):1699–1701.
107.
SilvaSJozwiakMTeboulJ-LPersichiniRRichardCMonnetX. End-expiratory occlusion test predicts preload responsiveness independently of positive end-expiratory pressure during acute respiratory distress syndrome. Crit Care Med. 2013;41(7):1692–1701.
108.
GuinotPGGodartJDe BrocaBBernardELorneEDupontH. End-expiratory occlusion manoeuvre does not accurately predict fluid responsiveness in the operating theatre. Br J Anaesth. 2014;112(6):1050–1054.
109.
RutlenDLWackersFJZaretBL. Radionuclide assessment of peripheral intravascular capacity: a technique to measure intravascular volume changes in the capacitance circulation in man. Circulation. 1981;64(1):146–152.
110.
JabotJTeboulJ-LRichardCMonnetX. Passive leg raising for predicting fluid responsiveness: importance of the postural change. Intensive Care Med. 2009;35(1):85–90.
111.
BoulainTAchardJTeboulJRichardCPerrotinDGiniesG. Changes in BP induced by passive leg raising predict response to fluid loading in critically ill patients. Chest. 2002;121(4):1245–1252.
112.
LamiaBOchagaviaAMonnetXChemlaDRichardCTeboulJ. Echocardiographic prediction of volume responsiveness in critically ill patients with spontaneously breathing activity. Intensive Care Med. 2007;33(7):1125–1132.
113.
PréauSSaulnierFDewavrinFDurocherAChagnonJ. Passive leg raising is predictive of fluid responsiveness in spontaneously breathing patients with severe sepsis or acute pancreatitis. Crit Care Med. 2010;38(3):819–825.
114.
MonnetXChemlaDOsmanD, et al.Measuring aortic diameter improves accuracy of esophageal Doppler in assessing fluid responsiveness. Crit Care Med. 2007;35(2):477–482.
115.
Alvarado-sánchezJI. Prueba de elevación de piernas pasiva. Rev Colomb Anestesiol. 2015;3(24):214–218.
116.
RehbergSErtmerCWestphalM. Valsalva, valsalva, may you give me a clue, who needs fluids in my ICU?Intensive Care Med. 2009;35(1):7–8.
117.
MonnetXBatailleAMagalhaesE, et al.End-tidal carbon dioxide is better than arterial pressure for predicting volume responsiveness by the passive leg raising test. Intensive Care Med. 2013;39(1):93–100.
118.
GuinotP-GBernardEDeleporteKPetiotSDupontHLorneE. Mini-fluid challenge can predict arterial pressure response to volume expansion in spontaneously breathing patients under spinal anaesthesia. Anaesth Crit Care Pain Med. 2015;34(6):333–337.
119.
GuinotP-GBernardEDefrancqF, et al.Mini-fluid challenge predicts fluid responsiveness during spontaneous breathing under spinal anaesthesia: an observational study. Eur J Anaesthesiol. 2015;32(9):645–649.
120.
Xiao-TingWHuaZDa-WeiL, et al.Changes in end-tidal CO2 could predict fluid responsiveness in the passive leg raising test but not in the mini-fluid challenge test: a prospective and observational study. J Crit Care. 2015;30(5):1061–1066.
121.
MallatJ. A comment on “changes in end-tidal CO2 could predict fluid responsiveness in the passive leg raising test but not in the mini-fluid challenge test: a prospective and observational study.”J Crit Care. 2016;31(1):273.
122.
MullerLCandelaDNyonzymaL, et al.Disagreement between pulse contour analysis and transpulmonary thermodilution for cardiac output monitoring after routine therapeutic interventions in ICU patients with acute circulatory failure. Eur J Anaesthesiol. 2011;28(9):664–669.
123.
MallatJMeddourMDurvilleE, et al.Decrease in pulse pressure and stroke volume variations after mini-fluid challenge accurately predicts fluid responsiveness. Br J Anaesth. 2015;115(3):449–456.
124.
RiversENguyenBHavstadS, et al.Early goal-directed therapy in the treatment of severe sepsis and septic shock. N Engl J Med. 2001;345(19):1368–1377.
125.
PinskyMR. Defining the boundaries of bedside pulse contour analysis: dynamic arterial elastance. Crit Care. 2011;15(1):120.
126.
PinskyMR. Functional hemodynamic monitoring. In: PinskyMRBrochardLManceboJAntonelliM, eds. Applied Physiology in Intensive Care Medicine 2: Physiological Reviews and Editorials; 2012:393.
127.
Monge GarcíaMIGil CanoAGracia RomeroM. Dynamic arterial elastance to predict arterial pressure response to volume loading in preload-dependent patients. Crit Care. 2011;15(1):R15.
128.
Monge GarcíaMGracia RomeroMGil CanoA, et al.Dynamic arterial elastance as a predictor of arterial pressure response to fluid administration: a validation study. Crit Care. 2014;18(6):626.
129.
CecconiMMonge GarcíaMIGracia RomeroM, et al.The use of pulse pressure variation and stroke volume variation in spontaneously breathing patients to assess dynamic arterial elastance and to predict arterial pressure response to fluid administration. Anesth Analg. 2015;120(1):76–84.
130.
GuinotP-GBernardELevrardMDupontHLorneE. Dynamic arterial elastance predicts mean arterial pressure decrease associated with decreasing norepinephrine dosage in septic shock. Crit Care. 2015;19(1):1–7.
131.
VosJJKalmarFStruysMMRFWietaschJKGHendriksHGDScheerenTWL. Comparison of arterial pressure and plethysmographic waveform-based dynamic preload variables in assessing fluid responsiveness and dynamic arterial tone in patients undergoing major hepatic resection. Br J Anaesth. 2013;110(6):940–946.
