Abstract
Summary
Coagulating lesions in posterior portion of midbrain and level of rostral pons of female rats resulted in increased corticosterone secretion into adrenal venous effluent in response to surgical stress. Lesions in posterior diencephalon and rostral midbrain structures resulted in depressed corticosterone secretion. Adrenal ascorbic acid response to stress was not correlated with corticosterone response. Ovulation was blocked in rats with brain lesions in any of these areas where concomitant mammillary peduncle damage was present.
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