Abstract
Summary
Isohydric alteration of the blood bicarbonate concentration in normokalemic, chronically hypokalemic, and acutely hyperkalemic rats did not affect blood citrate concentration. However, both renal citrate concent and urinary citrate excretion increased in the elevated bicarbonate groups at all potassium levels. Thus, bicarbonate, independent of its effect on pH, affects renal citrate metabolism regardless of the serum potassium level.
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